平時零散的蝗蟲可以吃沒問題，切記，蝗災的蝗蟲不能吃！
蝗蟲一旦聚集起來以後，因為群體目標太大容易被鳥類等天敵攻擊，所以他們會分泌PAN（苯乙腈）來釋放出難聞的氣味，讓天敵知難而退，如果這些鳥類還不退卻的話，他們就會把PAN經過一系列的反應變成HCN，對，就是氫氰酸，你們考慮下，劇毒。
中科院動物研究所去年1月在science advances發了一篇論文，主要是講蝗蟲的一種防禦天敵的機制：
文章裡講了一大堆化學反應和激素變化以及群體感應啥的，但這都不是重點。重點是這裡。
Our results indicate that PAN does not play a role in the aggregation of the migratory locust in the nymphal stage. In gregarious locusts, the PAN and HCN biosynthetic pathways participate in an antipredator defense mechanism that involves the coordination of olfactory aposematism and chemical defense (Fig. 5). The activation of a crucial CYP gene in the phenylalanine metabolic pathway during locust aggregation is an important mechanism in the adaptation of gregarious locusts to the group-living environment. There are many insectivorous bird species including seasonal migratory and nonmigratory birds in the field, so the aggregation exposures of the locusts obviously have the higher risks of predations. Because the activation of the PAN biosynthesis relies on the high population density of the locusts, the chemical defense of the locusts aims at the general predators in spite of the great tit being used in the behavioral tests. Thus, the plasticity of PAN biosynthesis in response to population density is crucial for the optimization of the antipredator defense strategies of locusts under diverse environmental conditions.
切記，蝗災的蝗蟲不能吃！
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